At this point, I allow myself to interpret and describe my own hypothesis regarding the identification of the real causes of the formation of “incurable” disease processes that irreversibly disrupt the work of the brain and the entire nervous system.
For many generations we have been widely informed that the human brain needs glucose as its best energy source to perform its work. From this we can draw the wrong conclusion that the brain does not need another energy sources, e.g. in the form of fat, because, as it is generally said, it encounters the cerebrovascular barrier on its way to the brain mass. Here, in my opinion, the tragic misconception is made that the supply of food components to them is carried out by the circulatory system ONLY. The mere claim that nerve cells take up the entire volume of the brain is wrong. Apart from them, as previously mentioned, there are various other types of cells, especially for the purpose of isolating one nerve from another, but also for structural purposes that maintain the optimal development of the central nervous system in the human body. If this were to be the case, as we have been taught for decades, that the brain is sufficient to supply about 140 grams of glucose per day for its proper functioning, then the question may be asked: how are all other types of cells present in the central nervous system supplied? It is definitely not done with glucose, because glial cells, e.g. Schwann cells, have a distinct character of fat cells. But after all, fats are not delivered to the brain cells in the blood.
We also need to ask ourselves another important question, how long do these non-nervous types of cells that fill our brains live? Only nerve cells are known not to reproduce by division, but to enlarge their structure by the growth of their new projections and axons. Could such insulating-structural Schwann cells wrapping each healthy axon exist and live as long as the nerve cell which they wrap and isolate? This is, of course, a false assumption, about which there is also no further information in the available literature. The results of scientific research on the actual causes of multiple sclerosis disease show that the reason of this incurable disease of the nervous system is the destruction of the fat tracts of Schwann cells. In this way, fragments of nerve axons are partially exposed, causing direct contact of the axon with the water environment, causing short circuits, “electric signal scattering” in the aquatic environment of the axon and, in total, false signal transmission in the nerve. There is a disease degeneration of Schwann cells, preventing the proper reaction of the nerve controlling the peripheral motor organs, such as moving the limbs or the functioning of glands, etc.
Why are there such deficiencies in the fat structure of Schwann cells? The answer must be sought in the fact that the most likely cause of the formation of “breaks” in the chain of Schwann cells strung on the axons is their dying due to the insufficient supply of dietary fats necessary for their proper functioning and reconstruction. At this point, we are confronted with another unexplained problem. After all, the modern canons of proper nutrition do not predict the need of brain for fats and the generally applicable dogma is to avoid dietary fats, especially of animal origin. Fat must therefore be delivered to the central nervous system and the brain via a different route than through the circulatory system. The only such known system of fat transport is the lymphatic system, but there are no typical lymphatic vessels in the brain for such transport to be carried out and logically explain.
Remember, however, that apart from our main lymphatic system in the human body, there is a smaller analogue in the brain and central nervous system called the glymphatic system. As it turns out both of these systems are anatomically and physiologically connected to each other. Information on this subject is not available in the available sources and it would be necessary to go deeper into this topic from the scientific point of view, which is still waiting for a wider discussion. We must be patient while looking for reliable information in this field. In such a case, we may dare to put forward another hypothesis that it is through the glymphatic system that systematic “deliveries” of fats necessary for the renewal and functioning of the brain take place in a lipoprotein appropriate for the glymphatic system, the name of which I have not yet been able to identify.